Conclusion


Jesus was a historical figure of great significance. It is clear that he had a profound effect on his original followers.  Jesus’ disciples would not recant their belief that Jesus was God’s messenger, the Messiah of Hebrew prophetic literature, in spite of torture and threats of death themselves. The biblical descriptions of Jesus’ crucifixion, by medically uneducated observers, correlates with modern medical understanding and experience. It is most logical medically that he died from the effects of traumatic shock, i.e. significant bodily injury with resultant critical blood volume depletion. The shock complication of an acute trauma-induced coagulopathy is reasonably considered as a possible contributor to Jesus’ rapid death.

The hypothesis that trauma-induced coagulopathy was a contributing factor of Jesus’ death is based on a forensic reconstruction from the biblical record. Specifically, the causative factors of trauma-induced coagulopathy appear to have been present based on the biblical record. Furthermore, this hypothesis has merit due to explanatory power with respect to Jesus’ unusually rapid death, as well as the observation of blood flow from Jesus’ corpse. Traumatic shock, complicated by trauma-induced coagulopathy, would lead to rapid death from blood volume depletion and a progressive loss of Jesus’ capacity to stop bleeding. In common vernacular, Jesus bled to death.

Jesus’ death as a result of shock and trauma-induced coagulopathy is also consistent with Jesus’ own statements the night prior to his execution.  It is, therefore, logical from both medical science and theological standpoints.

At Jesus’ last supper, the Passover meal prior to his crucifixion, he took the Passover cup and said, “This is my blood of the new covenant, which is poured out for many for the forgiveness of sins” (Matt 26:28).

From an orthodox Christian perspective, forgiveness comes through acceptance and belief in the vicarious atonement of Jesus’ crucifixion.